Publikationen (14) Publikationen von JAIONE AUZMENDI IRIARTE

2022

  1. Author Correction: PR-LncRNA signature regulates glioma cell activity through expression of SOX factors (Scientific Reports, (2018), 8, 1, (12746), 10.1038/s41598-018-30836-5)

    Scientific Reports

  2. Chaperone-Mediated Autophagy Controls Proteomic and Transcriptomic Pathways to Maintain Glioma Stem Cell Activity

    Cancer Research, Vol. 82, Núm. 7, pp. 1283-1297

  3. High levels of HDAC expression correlate with microglial aging

    Expert Opinion on Therapeutic Targets, Vol. 26, Núm. 10, pp. 911-922

  4. Intrinsic role of chaperone-mediated autophagy in cancer stem cell maintenance

    Autophagy, Vol. 18, Núm. 12, pp. 3035-3036

2021

  1. CRISPR/Cas9 deletion of SOX2 regulatory region 2 (SRR2) decreases SOX2 malignant activity in glioblastoma

    Cancers, Vol. 13, Núm. 7

  2. Identification of a dexamethasone mediated radioprotection mechanism reveals new therapeutic vulnerabilities in glioblastoma

    Cancers, Vol. 13, Núm. 2, pp. 1-23

2020

  1. Characterization of a new small-molecule inhibitor of HDAC6 in glioblastoma

    Cell Death and Disease, Vol. 11, Núm. 6

  2. Impact of Chaperone-Mediated Autophagy in Brain Aging: Neurodegenerative Diseases and Glioblastoma

    Frontiers in Aging Neuroscience, Vol. 12

  3. Methylation of MGMT promoter does not predict response to temozolomide in patients with glioblastoma in Donostia Hospital

    Scientific Reports, Vol. 10, Núm. 1

2019

  1. Liquid biopsy in glioblastoma: Opportunities, applications and challenges

    Cancers, Vol. 11, Núm. 7

  2. SIX1 represses senescence and promotes SOX2-mediated cellular plasticity during tumorigenesis

    Scientific Reports, Vol. 9, Núm. 1

2018

  1. Integrin α7: A novel promising target in glioblastoma stem cells

    Stem Cell Investigation, Vol. 5, Núm. 1

  2. PR-LncRNA signature regulates glioma cell activity through expression of SOX factors

    Scientific Reports, Vol. 8, Núm. 1

2017

  1. High expression of MKP1/DUSP1 counteracts glioma stem cell activity and mediates HDAC inhibitor response

    Oncogenesis, Vol. 6, Núm. 12